Written By Kami Ann Davis 🌹🍃
Don’t condemn my plant to promote your opioid, your chemical drug. The plant Kratom, in which I and millions of others consume, is derived from a COFFEE TREE. Suboxone is DERIVED FROM THE POPPY FLOWER. Here is information, an excerpt from Wikipedia and science direct to prove my point ~
But first, let me explain to you a little bit more about this horrifying substance. I used to support it more so, however; after researching it for the past 3 years, I have come to find that I cannot actually FIND very many positives about it. Yes, I guess it is better than heroin and it can save a life, however; there is not one person I’ve talked to that’s on it who reports they can get off of it. <<<<<This has nothing to do with transitioning.>>>>> OR, they are taking it only to report a slew of negative side effects.
Kratom, on the other hand.. has very little negative side effects. Kratom deserves to be recognized for all the good that it is DOING. But, it’s NOT.
Due to money hungry people, such as doctors and politicians, Kratom continues to be demonized and dismissed, as either harmful or having no therapeutic value, whatsoever.
That’s why I’m writing this article, so people out in the world can see there is another option besides Suboxone, or MAT (medicated assisted therapy). I do not condemn anyone having to take something on a regular basis permanently, however; I don’t support it, if it has a lot of negative side effects.
Being dependent on Suboxone is drastically different than being dependent on Kratom. Even though I haven’t actually taken Suboxone, I’ve spoken to hundreds who are taking it and say they are extremely addicted physically, emotionally, and mentally. With Kratom, it’s not like that.
There are so many differences between Kratom and Suboxone. Kratom does not grip your psyche like Suboxone does, or other opioids or psychotropics, for that matter. Kratom doesn’t produce horrific withdrawals like Suboxone, nor does it have negative side effects, such as nodding off in the middle of activities. Kratom isn’t highly addictive like Suboxone, either.
Let’s talk about derivatives now. Kratom is DERIVED FROM THE COFFEE TREE, the rubiaceae. Suboxone is derived from the poppy flower. Even worse, it IS a chemical drug that’s been synthesized in a lab out of thebaine. Thebaine is an alkaloid from the poppy flower.
This is very important to understand because unless you have a pharmacological background such as I, you may be confused in understanding medications and how they’re derived and made. It’s crucial that you start learning this though, because otherwise you end up being at the mercy of others, like your doctors and our government. That’s not always your safest bet.
There is a huge difference as to why Kratom isn’t highly addictive like OPIOID DERIVATIVES, such as Suboxone and Methadone. It is because Kratom contains alkaloids that are solely derived from a coffee tree plant. Suboxone is derived from an alkaloid (thebaine) that is derived from the poppy flower. Kratom is NOT. This is why Kratom is such a healthier and SAFER option, when people are trying to get clean and free from their addictions.
Before, you skeptics start saying Kratom is addictive too. Let me stop you right there and CORRECT your way of thinking. There ARE safe addictions, as opposed to harmful addictions. Any addiction that produces harm to an individual or their surroundings has a right to be condemned. However, if it is a minority then one needs to be reasonable and rational and look at the bigger picture. Because if we used the rationale that if any medication produced ANY negative side effect that we ban it, we wouldn’t have any medication or treatment left to take.
Kratom doesn’t cause deadly withdrawals or harmful side effects. If you are a harmful person to begin with though and you start consuming Kratom, I guess you could be harmful in taking too much and perhaps experience nausea or negative effects. But, even still ..Kratom doesn’t cause death like opioid derivatives do. So, you cannot lump this plant in the same category as an actual opioid derivative. It’s not derived from any opioid plant, nor does it bind in the same way. It’s not even chemically made up the same way.
Science that backs up the safety of Kratom ~ https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5813050/
Kratom ~ Binding only partially to the receptors in the brain, thus zero risk for respiratory depression ~ https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5344672/
Here is the science and chemical makeup of Suboxone ~
‘Buprenorphine, a synthetic opioid, treats pain and opioid addiction. It underwent development in the late 1960s. It a synthetic analog of thebaine, an alkaloid compound derived from the poppy flower. It is a schedule III drug, which means that it has some potential for moderate or low physical dependence or high psychological dependence.
Buprenorphine is FDA-approved for acute pain, chronic pain, and opioid dependence. It is an agent used in agonist substitution treatment, which is a process for treating addiction through the use of a substance (such as buprenorphine or methadone) to substitute for a stronger full agonist opioid (such as heroin). The prescriber then tapers down the substitute, and the patient withdraws from the opiate addiction with minimal discomfort. Buprenorphine substitute treatment allows the patient to focus on therapy instead of uncomfortable withdrawals. It is an effective option to treat opioid dependence and reduces cravings and improves the quality of life for patients undergoing addiction treatment. It allows the patient to circumvent many of the uncomfortable symptoms of opioid withdrawal, creating a treatment plan that patients are more likely to adhere to, thereby decreasing reducing morbidity and mortality.’
Buprenorphine is a thebaine derivative, and its analgesic effect is due to partial agonist activity at μ-opioid receptors, i.e. when the molecule binds to a receptor, it is only partially activated in contrast to a full agonist such as morphine. Buprenorphine also has very high binding affinity for the μ receptor such that opioid receptor antagonists (e.g. naloxone) only partially reverse its effects. Buprenorphine is administered via a transdermal route in 35, 52.5 and 70 mcg/hour patches that deliver the dose over 96 hours. It is not administered orally, due to very high first-pass metabolism. Buprenorphine is metabolised
USES: This medication contains 2 medicines: buprenorphine and naloxone. It is used to treat narcotic (opioid) dependence/addiction.
<<<<Buprenorphine belongs to a class of drugs called mixed narcotic agonist-antagonists. >>>>
Buprenorphine helps prevent withdrawal symptoms caused by stopping other opiate-type narcotics.Naloxone is a narcotic antagonist that blocks the effect of narcotics and can cause severe narcotic withdrawal when injected. It has little effect when taken by mouth or dissolved under the tongue. It is combined with buprenorphine to prevent abuse and misuse (injection) of this medication. This combination medication is used as part of a complete treatment program for drug abuse (such as compliance monitoring, counseling, behavioral contract, lifestyle changes).
William Charles Evans BPharm BSc PhD DSc FIBiol FLS FRPharmS, … Daphne Evans BA MA, in Trease and Evans’ Pharmacognosy (Sixteenth Edition), 2009
Thebaine is the predominant alkaloid of this species and in a UN-backed programme of large-scale cultivation trials were organized in various countries. High yielding strains were introduced, for example, Ayra II, a race obtained from west Iran in 1974 which gives 3.5% thebaine in the dried capsules. Problems associated with the development were the insufficiency of seed of high-yielding strains and the poorer crops obtained when the plants were removed from their normal environment. However, political decisions also jeopardized the continuation of the programme.
P. bracteatum produces some 27 alkaloids belonging to 10 of the 14 alkaloid groups described for Papaver. The biogenesis of thebaine follows the same pathway as in P. somniferum. Feeding experiments with labelled intermediates have shown that the plant is capable of converting codeinone to codeine but cannot perform either of the demethylations leading to codeine or directly to morphine. Thebaine does not appear to be entirely an end-product and undergoes further metabolism to unknown products (for a report, see H. G. Theuns et al., Phytochemistry, 1985, 24, 581). A more recently described new alkaloid is salutaridine-N-oxide (G. Sariyar et al., Planta Med., 1992, 58, 368).
Pharmacological treatments in rheumatic diseases
Benazir Saleem MBChB MRCP, Philip G. Conaghan MBBS PhD FRACP FRCP, in Rheumatology, 2010
Buprenorphine is a thebaine derivative, and its analgesic effect is due to partial agonist activity at μ-opioid receptors, i.e. when the molecule binds to a receptor, it is only partially activated in contrast to a full agonist such as morphine. Buprenorphine also has very high binding affinity for the μ receptor such that opioid receptor antagonists (e.g. naloxone) only partially reverse its effects. Buprenorphine is administered via a transdermal route in 35, 52.5 and 70 mcg/hour patches that deliver the dose over 96 hours. It is not administered orally, due to very high first-pass metabolism. Buprenorphine is metabolised by the liver and the metabolites are eliminated mainly through excretion into bile. The elimination half-life of buprenorphine is 20–73 hours (mean 37). Common adverse drug reactions associated with the use of buprenorphine are similar to those of other opioids and include: nausea and vomiting, drowsiness, dizziness, headache, itch, dry mouth, miosis, orthostatic hypotension, male ejaculatory difficulty, decreased libido and urinary retention. Constipation and central nervous system (CNS) effects are seen less frequently than with morphine. Hepatic necrosis and hepatitis with jaundice have been reported with the use of buprenorphine, especially after intravenous injection of crushed tablets.’
No wonder Suboxone is so horrible… look at the class of drugs they fall under ~ Mixed narcotic agonist antagonists.
In case some don’t understand Brand versus generic, regarding medications, it means that a lot of drugs may have two names. For example, acetaminophen is the generic name for Tylenol. ‘Tylenol’ is the brand name. Or ..like buprenorphine is the generic name, which is the drug itself and Suboxone is the brand name but it doesn’t mean it has different ingredients always and that’s why it’s important to learn and do research, if you don’t have nursing and pharmacologic background.
Again… Suboxone USES: This medication contains 2 medicines: buprenorphine and naloxone. It is used to treat narcotic (opioid) dependence/addiction. Buprenorphine belongs to a class of drugs called mixed narcotic agonist-antagonists. Buprenorphine helps prevent withdrawal symptoms caused by stopping other opiate-type narcotics.Naloxone is a narcotic antagonist that blocks the effect of narcotics and can cause severe narcotic withdrawal when injected. It has little effect when taken by mouth or dissolved under the tongue. It is combined with buprenorphine to prevent abuse and misuse (injection) of this medication. This combination medication is used as part of a complete treatment program for drug abuse (such as compliance monitoring, counseling, behavioral contract, lifestyle changes).
Kratom deserves to be made known to every single person on this planet. If someone would rather take Suboxone, so be it. But, at least let the person have the option and free choice to choose. Keeping people in the dark is the same thing as lying. Withholding information <<<knowingly>>> is the same as omitting the TRUTH. Omitting the truth is LYING. Don’t abuse your position of power. I am talking to YOU DOCTORS and POLITICIANS. Stop choosing one side over the other. Or at least let the people make that decision for THEMSELVES.
Written By Kami Ann Davis 🌹🍃